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Children's fructose intolerance

Children's fructose intolerance


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Hereditary fructose intolerance (HFI) in children is a disease caused by the absence of an enzyme that transforms the sugar contained in fruit and honey.

This genetic alteration prevents fructose from being synthesized in the proper way for the child's body, accumulating in the liver, kidneys or intestine.

Fructose or levulose is the type of sugar that both fruit and honey contain. While table sugar is formed from the union of glucose and fructose, fructose itself is a monosaccharide made up of a single link.

This means that it does not need to be digested and can pass directly into the bloodstream from the intestine, to finish its process in the liver, where it is transformed into fructose 1 phosphate, a new substance on which the enzyme aldolase B acts, responsible from the transformation of fructose to produce, for example, energy.

The first symptoms that appear when a child is intolerant to fructose, after consuming a food with this type of sugar, are abdominal pain and vomiting that can end up leading to severe hypoglycemia.

Treatment is basically preventive. The child's diet should be controlled to avoid the intake of fruit or honey. In addition, other types of sugars such as sucrose or sorbitol should be avoided.

But by eradicating the fruit from the child's diet, the greatest source of vitamin C, necessary for proper child development, is also being suppressed, so it is recommended to supplement the absence of this nutrient.

Fructose malabsorption is a more common disease than hereditary fructose intolerance, occurring in approximately 30% of the world's population. In this case, it is the intestinal cells that are not capable of fully absorbing fructose, causing the child to have abdominal pain, gas, diarrhea or nausea.

Diego Fernandez. Editor of our site

You can read more articles similar to Children's fructose intolerance, in the category of Mental Disorders on site.


Video: Hereditary Fructose Intolerance Mnemonic for the USMLE (May 2022).